Do you have patients suffering from infections that die for some time and then come back? Bacterial and infectious biofilms can be the reason. Learn more about biofilms and their treatment.
Most bacteria are present in biofilms, not as single-acting cells.
The famous picture of bacteria shows individual cells that roam and excrete toxins and damage the host. In this way, most bacteria do not have this planktonic structure in the human body, but in small networks called biofilms. To form a biofilm, the bacteria first bind quickly to the surface and then build a polysaccharide structure that also links to magnesium, calcium iron, or another mineral.
What is an anti biofilm supplement?
Bacteria, yeasts, and a few different parasites and different microorganisms generally work without reservation as individual cells in the so-called plankton structure. When these individual microorganisms are bound to a surface, for example, the gastrointestinal tract, the group in a network and form a state, larger provinces can have different types, which makes structures very complicated.
Biofilms are a typical component of bacterial infections that can cause real conditions, e.g., B. Candidiasis, skin problems, pneumonia, and various levels of poison. This is just the tip of the iceberg. The standard treatment of biofilms has little effect on the microorganisms it contains. Biofilm X is tailored to improve the body's ability to separate and dispose of biofilms.
This network forms a kind of protective shield around the group, making the microorganisms that cover it inside difficult to identify and manipulate during testing.
Studies have shown that severe infections are often acceptable to include planktonic types of parasites, bacteria, and yeast - single free cells - that can usually be treated with anti-infectives / antimicrobials. However, when these microorganisms form a biological framework, the infection becomes continuous. It is incredibly insensitive to antimicrobial agents or other antimicrobial agents and unique body guarantees - the resistant tires.
In addition to growth, there is at least one type of bacteria in the biofilm film that shares nutritional supplements and DNA and tests changes to avoid the safe framework. Because the biofilm requires less oxygen and fewer supplements and regulates the pH in the center, it is a threat network for most anti-infectives. The biofilm also forms physical boundaries that protect most endangered cells from the identification of pathogenic bacteria.
The current care model misses the mark.
The current care model usually predicts a severe infection caused by plankton bacteria. However, since most bacteria are contained in the biological membranes, human service providers reward most diseases ineffectively. According to the National Institutes of Health, over 80 percent of human bacterial infections are related to bacterial biology. While planktonic bacteria can be safe from infection by qualitative shifts, biofilm is often safe from infection for several reasons - physical, composite, and genetic. Rewarding diseases related to biofilms that use antimicrobial agents is a difficult task. For example, anti-infectious agents appear to initially work in patients with inflammatory bowel inflammation, followed by a "rebound" in which the signs break out again, apparently due to bacteria avoiding the anti-infectious agent in a biofilm film.
Biofilms are hidden in the passages of the nose and digestive system.
Biofilms are essential topics connected with endoscopy methods, vascular connections, clinical and dental prosthetic inputs, and severe skin injuries. Biofilms found along the epithelial coatings of the nasal passages and the digestive system are less well known.
The digestive system is an ideal prerequisite for bacteria, parasites, and related biofilms given the large surface and the constant immersion of food supplements. As insurance, the gastrointestinal epithelium is fixed with flexible, sticky body fluids, but it can be damaged very well in patients with higher irritation, IBD, and various diseases. This opens the door so that bacteria can stick to the surface and develop biofilms. The epithelium with which it is connected and damaged is regularly adjusted.
Biofilms are difficult to diagnose
Several problems make it challenging to identify biofilms.
First, the bacteria are hidden in the biofilm in the frame. Therefore smears and societies often appear negative. Stool examinations often do not contain any bacteria in the biofilms.
Second, it isn't easy to get biofilm tests in the digestive system. This technology requires an internal and optical endoscope in which the biofilm is located. Likewise, there is currently no system for flushing out biofilms from the gastrointestinal tract.
Third, biofilm bacteria are not natural to revise. Regardless of whether you can get an example, the test can be negative again due to the less changed need for microorganisms, which invalidates standard culture procedures.
Fourth, biofilms can perform a function in the solid intestine, making it challenging to identify pathogenic and acoustic networks.
Although culture can passively return, living organisms in biofilms can get rid of toxins that cause disease. Some doctors look for mycotoxins in the urine to identify biofilms, but I am not yet impressed by the study behind them. Because bacteria retain minerals from the host, it is believed that the lack of minerals is related to the proximity of the biofilms. However, mineral deficiencies are so significant for everyone to use as a guideline.
Biofilms are not visible to many diseases.
The clinical network is gradually managing a safe antibacterial infection, and it turns out that the biofilm works outside the camera:
Up to 33% of patients with a streptococcal sore throat, which are regularly carried out by purulent genes, do not interact with antioxidants. In a survey, each of the 99 bacteria that cause a sore throat isolated the biofilms formed.
Ten to 20 percent of Burgdorferi-infected people who are contaminated with Lyme disease have outlined manifestations, possibly due to an antimicrobial blockage and proximity to the biofilms.
Lupus is stimulated by infection, exacerbation, or injury. In this disease of the immune system, the destruction of the cell by NETosis instead of apoptosis transforms the immunological framework against itself. It is believed that it contains biofilms.
Regarding persistent sinusitis (CRS), "the antibacterial or antifungal agents showed no advantage over counterfeit treatment in controlled, irregular filtrations." Bacterial and parasitic biofilms can be found reliably in the nasal passages of these patients.
Colorectal cancer (IBD) antitoxin treatment can work for a while, but the most common seizures persist throughout the patient's life. Biofilms have been associated with Crohn's disease and ulcerative colitis.
Also, the biofilms were involved in persistent ear infections, persistent weakness, stiffness, and heartburn.
The most effective way to treat biofilms
Antimicrobial after IBD antioxidant. Corticosteroids for CRS. When the live film is busy, these standard "treatments" don't help. Instead, doctors have to separate the biofilms, attack the pathogenic bacteria inside, and wipe off the additional network, DNA, and minerals.
The imbalance of the biofilm is the primary strategy. Proteins, such as nattokinase and lumbrokinase, have been used extensively as an input coating to combat biofilms. The Cohen protocol suggests much of the 50 mg case of nattokinase and half of the 20 mg case of lumbrokinase for young adults with persistent sore throats and chemical imbalance.
Lauricidin (different structures: monolaurin, lauric erosion, and glycerol monolaurate) is a natural surfactant in coconut oil that limits the improvement of biofilms. In my training, I also use it as an opportunity for a gentler antimicrobial specialist.
Colloidal silver convinces with the reward of topical biofilms, for example, in wound dressings. In vivo applications are still under investigation. Although colloidal silver is used effectively to treat the sheep model of bacterial sinusitis, it has not shown similar viability in some primer people.
Biofilm offers a protective shield against discovery and a certain degree of security against treatment. Anti-infectious and antimicrobial agents can survive with longer victims without many plankton bacteria. However, it can be annoying to get to those in the biofilm because the antimicrobial/antimicrobial agents cannot infiltrate the biofilm.
Also, however, it has been recommended that any antimicrobial progress within the biofilm, as opposed to thwarting the arrangement of the biofilms, actually increases. These are just a few of the many variables that contribute to the recent improvement in the antitoxic blockade.
For example, because the development of biofilms, stationary parasites, bacteria, and organisms is structured in such a way that Candida albicans appear to be connected to the hip, scientists accept that anti-infectious / antimicrobial therapy mixed with biofilm cucumbers can be a response to efforts. Infections. This agrees positively with our preoccupation with clinical practice and is the reason why biofilm dysfunction should be considered as a critical aspect of a long-term IBS treatment protocol.
A biofilm infection is an invisible piece of persistent digestive inflammation. In addition to the fact that they carry out an infection identification test, they convince the treatment with common strategies that are practically unthinkable. At least not anymore. We are sure that you have found this blog to be supportive, and make sure that each of your future protocols has an apparent cause of a biofilm defect to provide the most obvious way to restore GI.